OS - ECG from pathophysiology to clinical interpretation - DVA01064

• Title: OS - ECG from pathophysiology to clinical interpretation
• Guaranteed by: Department of Pathophysiology (13-312)
• Faculty: Second Faculty of Medicine
• Actual: from 2024
• Semester: summer
• Points: 3
• E-Credits: 3
• Examination process: summer s.:
• Hours per week, examination: summer s.:0/20, C [HS]
• Capacity: unknown / 32 (32)
• Min. number of students: 10
• 4EU+: no
• Virtual mobility / capacity: no
• State of the course: taught
• Language: English
• Teaching methods: full-time
• Note: course can be enrolled in outside the study plan; enabled for web enrollment
• Guarantor: MUDr. Jan Doul, Ph.D.
• Teacher(s): MUDr. Jan Doul, Ph.D.

Course completion requirements

80% participation in the lectures.

In the case of absences due to justifiable reasons, substitution is made by the self-study of the lectures and interpretation of ECG during credit. 50% participation in classes is required. "Half" absences can be counted in case of non-participation in parts of lectures or practicals.

Last update: Procházková Helena, Bc. (30.08.2024)

Literature

Study materials on the Moodle

Last update: Procházková Helena, Bc. (30.08.2024)

Requirements to the exam

Course completed by credit without exam.

Last update: Procházková Helena, Bc. (30.08.2024)

Syllabus

The teaching takes place in the form of lectures followed by the description of specific ECG curves by the students.

Description of the ECG curve:

Basics of electrocardiography - origin of signal on ECG, ECG leads

Procedure for description of the ECG curve - rhythm, action - regular, irregular, frequency, electrical axis of the heart, transition zone

The formation of individual waves on the ECG - P wave, PQ interval, QRS complex, ST segment, T wave, U wave, QT and QTc interval

Dextrocardia, dextroposition of the heart, swapping of ECG leads

ECG manifestations of diseases:

Ischemic changes - ST elevation/depression (toombstoning), T inversion, Q waves, poor R wave progression, timecourse of the development of ischemic changes, localization of infarction according to the ECG

Left ventricular hypertrophy, right ventricular hypertrophy, ECG findings in pulmonary embolism

Pericarditis, pericardial effusion and tamponade, myxedema and other causes of low voltage, hypothermia, pacing, pediatric ECG

Hyperkalemia and hypokalemia, hypercalcemia and hypocalcemia, digoxin

Arrhythmias:

Conduction system, action potential of the myocardium and conduction system, sinus arrhythmia

Mechanisms of arrhythmias - reentry, ectopic formation, afterdepolarization, blockades

Pathological sources of rhythm - ectopic atrial, nodal, ventricular

SV tachycardia - sinus, atrial, multifocal atrial, atrial fibrillation, atrial flutter, AVNRT

Extrasystoles, Lown's classification of extrasystoles, R on T phenomenon

Ventricular tachycardia, ventricular fibrillation, asystole, pulseless electrical activity

Long QT syndrome and torsades de pointes

AV blocks - I degree, Mobitz I (Wenkebach) and II, III degree, SA blocks, sick sinus syndrome

RBBB; complete, incomplete; LBBB - normal image, hemiblocks

WPW syndrome and LGL syndrome

Principles of treatment of arrhythmias

Differential diagnosis:

ST elevations

Acute myocardial infarction with the blockade of the left and right bundle branch

Q Waves

Poor R wave progression in the chest leads

Dominant R wave in V1

Tachycardia: narrow vs wide QRS complex; regular vs irregular

Basic findings on a pediatric ECG

Practice:

12 lead ECG

Aplysia Cardiovascular Lab - model of the effect of arrhythmias on circulation

Arrhythmias and cardiopulmonary resuscitation

Ergometry

Last update: Procházková Helena, Bc. (30.08.2024)