velikost textu
Výsledky projektu Kooperace ARHGAP42 a PKN3 v nádorové signalizaci
Výsledky
  Typ výsledku
Autor celku
Název celku(Celkem 2 zázn.)
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| Dibus, Michal; Brábek, Jan; Rösel, Daniel. A Screen for PKN3 Substrates Reveals an Activating Phosphorylation of ARHGAP18. International Journal of Molecular Sciences, 2020, sv. 21, s. 1–17. ISSN 1422-0067. IF 4.556. [Článek v časopise] |
| Li Q, Dibus M, Casey A, Yee CSK, Vargas SO, Luo S, Rosen SM, Madden JA, Genetti CA, Brabek J, Brownstein CA, Kazerounian S, Raby BA, Schmitz-Abe K, Kennedy JC, Fishman MP, Mullen MP, Taylor JM, Rosel D, Agrawal PB. A homozygous stop-gain variant in ARHGAP42 is associated with childhood interstitial lung disease, systemic hypertension, and immunological findings. PLOS Genetics, 2021, sv. 17, s. 1–18. ISSN 1553-7404. IF 5.917. [Článek v časopise] The results of this project were published in Plos Genetics (Li Q, Dibus M, Casey A, et al. A homozygous stop-gain variant in ARHGAP42 is associated with childhood interstitial lung disease, systemic hypertension, and immunological findings. PLoS Genet. 2021;17(7):e1009639). The support of GAUK is in the paper acknowledged. |